human embryonic diploid lung fibroblast helf cell line human fetal lung fibroblast 1 (ATCC)
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human embryonic diploid lung fibroblast helf cell line human fetal lung fibroblast 1
Human Embryonic Diploid Lung Fibroblast Helf Cell Line Human Fetal Lung Fibroblast 1, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 643 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+embryonic+diploid+lung+fibroblast+helf+cell+line+human+fetal+lung+fibroblast+1/HFL1/pm38908195-97-8-26
Average 96 stars, based on 643 article reviews
Human Embryonic Diploid Lung Fibroblast Helf Cell Line Human Fetal Lung Fibroblast 1, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 643 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+embryonic+diploid+lung+fibroblast+helf+cell+line+human+fetal+lung+fibroblast+1/HFL1/pm38908195-97-8-26
Average 96 stars, based on 643 article reviews
human embryonic diploid lung fibroblast helf cell line human fetal lung fibroblast 1 - by Bioz Stars,
2026-10
96/100 stars
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other:Article Title: Role of TRIM59 in regulating PPM1A in the pathogenesis of silicosis and the intervention effect of tanshinone IIA. Article Snippet: This study examines the involvement of TRIM59 in silica-induced pulmonary fibrosis and explores the therapeutic efficacy of Tanshinone IIA (Tan IIA).. In vivo experiments conducted on rats with silica-induced pulmonary fibrosis unveiled an increase in TRIM59 levels and a decrease in PPM1A levels.. Subsequent investigations using in vitro silicosis cell models demonstrated that modulation of TRIM59 expression significantly impacts silicosis fibrosis, influencing the levels of PPM1A and activation of the Smad2/3 signaling pathway. |